JITC Digest July 2026

By JITC Publications posted an hour ago

  

INSIDE THIS ISSUE:  

Letter from the Editor| JITC  Editor Picks|New Special Series Myeloid Cells in Immuno-Oncology|JITC Meet-the-Editor Seession at JACI (Japan)|Popular Articles 

Letter from the Editor

Hello JITC Readers, 

This month I would like to call attention to the recently completed “Myeloid Cells in Immuno-Oncology” special series now out in JITC. We also have several exciting series in the works which will be appearing over the next few months, including ones on AI in IO and neurobiology/immunobiology intersections. 

 

Myeloid Cells in Cancer Immunology
Cancer in adults (but not children) most often arises in the setting of chronic inflammation with rare exception, most sarcomas and gliomas as two such exceptions. Following seven to ten years, nascent tumor cells arising in this setting associated with release of damage-associated molecular pattern (DAMP) molecules such as HMGB1, DNA itself, and the S100 family of molecules, recruiting and activating myeloid cells including neutrophils and circulating monocytes. These in turn play critical roles in the wound-healing process as well as modulating tissue resident memory T cells, deposited gamma delta T cells, and recruited T and NK cells. The ‘yin and yang’ of these myeloid cells, playing most often an inhibitory response to lymphoid cells, has now been more interestingly broadened to consider not only their intrinsic diversity but also their potential anti-tumor roles as illustrated by some of the papers in the new JITC series. We have still to fully discern whether cancer most often arises with a chaotic big bang or as stepwise accumulation of mutations as originally considered. Efforts to align with the biology in our therapeutics, consider how to integrate vascular biology and access to immune effectors, and preempt tumor development with immune restitution remain to be integrated in our full understanding to enable immune interception/prevention of cancer. 

 

30th Anniversary of the Japanese Association of Cancer Immunology (JACI 2026
I will be having the chance to visit Japan and greet my former postdoctoral fellow, now at Riken, Dr. Shin-ichiro Fujii. He and I will be at the upcoming 30th Annual JACI Meeting later this month, scheduled for July 22–24, at the Kagawa International Conference Hall and Sunport Hall in Takamatsu, Japan. 

 

Themed "Cancer vs. Immunity – Unraveling the Dynamics of the Battle," the event explores tumor microenvironments, antibody engineering, and immunotherapy resistance. For more specifics on the itinerary and abstracts, you can review the official schedule. In addition to a talk on tumor infiltrating lymphocytes, I will also be at a meet-the-editor session at the 30th JACI. See below for more details on this session.

 

Highlighted Manuscripts
Apropos the focus on neutrophil biology and inflammation, I have focused on those papers with a distinctly myeloid pitch. I encourage you to examine these and the others published in JITC this past month. The first article noted below from Taito Miyamoto and colleagues at the Wistar Institute proposes a rather intricate and interesting pathway in murine and human ovarian cancer studies that limits immune responses following chemotherapy. IL-1 provided by chemotherapy-stimulated tumor resident macrophages activates fibroblasts to release CXCL1, thereby recruiting neutrophils. Neutrophil extracellular traps (NETs) limit the effectiveness of T cells. Anna Rita Redavid and colleagues in Rome reviewed the extensive and largely descriptive but increasingly foundational studies focusing on NK cells interacting with tumor-associated macrophages (TAMs). How to promote and align tissue-resident and inflammatory macrophages with effector NK (and T) cells coordinately represent areas of considerable foment and interest. Sanxiu He and colleagues from Chongqing further advance the importance of TAMs, focusing on the ability of lymphoma cells to release the DAMP HMGB2 through exosomes, which, interestingly, upregulates TAM E3 ubiquitin ligase TRIM65, promoting degradation of the NLRP3 inflammasome and thereby limiting antitumor responses.

 

A broad look at questions arising in myeloid biology in cancer was highlighted by a special series coordinated by Drs. Jennifer L. Guerriero, Dmitry I. Gabrilovich, and Judith A. Varner. They focus on burning questions in myeloid biology in cancer and introduce the series along with an editorial. See here and the special feature below for additional information on the complete series.

 

Regards,

Michael T. Lotze, MD, FAIO
Editor-in-Chief

 Journal for ImmunoTherapy of Cancer 

JITC Editor Picks

Chemotherapy induces an IL1β-dependent neutrophil recruitment and activation that promote chemoresistance in metastatic ovarian cancer

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From the Authors – What this study adds:
  • The authors found that chemotherapy drives inflammatory reprogramming of the tumor microenvironment in ovarian cancer, with myeloid cell-derived interleukin 1 beta (IL1β) playing a central role in mediating chemoresistance. Following chemotherapy, IL1β promotes neutrophil recruitment, leading to neutrophil extracellular trap (NET) formation and altered T-cell responses that may contribute to chemoresistance. This study provides new experimental evidence for chemoresistance mechanisms in high-grade serous carcinoma (HGSC), whereby chemotherapy-induced inflammatory remodeling of the intraperitoneal tumor microenvironment impairs tumor sensitivity to chemotherapy.

Targeting macrophage-driven NK cell immunosuppression to improve cancer immunotherapy  

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From the Authors  – Abstract:
  • Natural killer (NK) cells are critical effectors of antitumor immunity, however, their cytotoxic function is frequently impaired within the tumor microenvironment (TME). Tumor-associated macrophages (TAMs), the most abundant immune stromal population in solid tumors, play a central role in shaping NK cell responses through a broad range of mechanisms, including receptor–ligand interactions, immunosuppressive cytokine signaling, metabolic reprogramming, and engagement of immune checkpoint pathways. Here, we review current insights into the bidirectional crosstalk between TAMs and NK cells and discuss therapeutic strategies aimed at restoring NK cell activity by targeting TAMs. These include macrophage depletion and reprogramming approaches, modulation of metabolic and transcriptional pathways, and interventions targeting cytokine networks and immune checkpoints. We further examine emerging strategies that reshape the TME to enhance NK-macrophage cooperation, such as induction of inflammatory cell death, modulation of innate immune signaling pathways, and the development of synthetic NK cell engagers. In addition, we highlight the impact of macrophage ontogeny, tissue residency, and spatial organization on NK cell function, emphasizing how distinct microanatomical niches within the TME regulate immune cell interactions and influence therapeutic responses. Finally, we summarize translational advances and ongoing clinical efforts aimed at integrating TAM-targeted therapies with NK cell-based approaches. Collectively, these findings provide a conceptual and mechanistic framework for the rational design of combination immunotherapies that leverage macrophage-NK cell interactions to enhance innate immune responses and improve cancer treatment outcomes.

Tumor-derived HMGB2 induces M2-like macrophage polarization via TRIM65-mediated NLRP3 degradation to promote DLBCL progression

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From the Authors – What this study adds:
  • This study reveals that diffuse large B-cell lymphoma (DLBCL) cells secrete HMGB2 primarily via exosomes, which upregulates the E3 ubiquitin ligase TRIM65 in macrophages, leading to ubiquitin-mediated degradation of the NOD-like receptor protein 3 (NLRP3) inflammasome.
  • This novel HMGB2-TRIM65-NLRP3 axis drives macrophages toward an immunosuppressive M2-like phenotype and impairs their phagocytic capacity, thereby promoting tumor progression and contributing to therapy resistance.

Expert opinion on burning questions in cancer myeloid cell biology

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From the Authors – Introduction:

  • As part of the special Journal for ImmunoTherapy of Cancer (JITC) series, “Myeloid Cells in Immuno-Oncology,” leading experts were asked to provide their perspectives and answer provocative questions on controversial and evolving areas in the field of myeloid biology. Their responses aim to further the discussion and help advance the biological understanding of myeloid cell biology within the tumor and strategies to modulate the function and activity of myeloid cells for anticancer therapy.

Special Series: Myeloid Cells in Immuno-Oncology

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The complete JITC special series, “Myeloid Cells in Immuno-Oncology,” is now available. This series features expert, in-depth reviews on the roles of myeloid cells and their metabolic regulation in cancer immunotherapy and presents multiple expert perspectives on controversial and evolving areas in the field of myeloid biology. 

 

Edited by Dmitry I. Gabrilovich, MD, PhD, Jennifer L. Guerriero, PhD, and Judith A. Varner, PhD, and developed in collaboration with The Myeloid Network, this special series builds on the momentum and enthusiasm generated by prior jointly sponsored SITC Annual Meeting pre-conference workshops and programs focused on elucidating the mechanisms and therapeutic targeting of myeloid cells in immuno-oncology. 

Meet-the-Editor Session at JACI

For those attending the 2026 Japanese Association of Cancer Immunology (JACI) Annual MeetingJITC Editor-in-Chief Dr. Michael Lotze is hosting a session, “How to Write a Great Paper and Get it Published” on July 23, 11 a.m.–12 p.m. in the Main Hall (Hall 1), Kagawa International Conference Hall, 6th Floor, Tower Building, Takamatsu, Japan.

 

Stop by to say hello to Dr. Lotze, gain valuable insights to maximize the impact of your next submission, and learn more about JITC.

Popular Archive Articles 

The selections below represent some of the most popular content published in JITC over the past two years. Explore additional thematic content in JITC 's Collections or access the rest of JITC 's archives for a look at all the journal has to offer.

Mitochondria-targeted photodynamic therapy triggers GSDME-mediated pyroptosis and sensitizes anti-PD-1 therapy in colorectal cancer (1 March 2024) 

POSTN+ cancer-associated fibroblasts determine the efficacy of immunotherapy in hepatocellular carcinoma  (27 July 2024) 

Metastatic gastric cancer target lesion complete response with Claudin18.2-CAR T cells  (5 February 2024) 

Severe acute myositis and myocarditis on initiation of 6-weekly pembrolizumab post-COVID-19 mRNA vaccination (24 April 2024) 

Four-year clinical update and treatment switching-adjusted outcomes with first-line nivolumab plus ipilimumab with chemotherapy for metastatic non-small cell lung cancer in the CheckMate 9LA randomized trial (12 February 2024) 

Phase 1a dose escalation study of ivonescimab (AK112/SMT112), an anti-PD-1/VEGF-A bispecific antibody, in patients with advanced solid tumors  (19 April 2024) 

Immunotherapy-based regimens for patients with EGFR-mutated non-small cell lung cancer who progressed on EGFR-TKI therapy  (16 April 2024) 

CCR5 and CCL5 in metastatic colorectal cancer (7 May 2024) 

Efficacy and safety of autologous tumor-infiltrating lymphocytes in recurrent or refractory ovarian cancer, colorectal cancer, and pancreatic ductal adenocarcinoma  (2 February 2024) 

Safety and biological outcomes following a phase 1 trial of GD2-specific CAR-T cells in patients with GD2-positive metastatic melanoma and other solid cancers (15 May 2024) 

CD276 regulates the immune escape of esophageal squamous cell carcinoma through CXCL1–CXCR2 induced NETs  (9 May 2024) 

An oncolytic vaccinia virus encoding hyaluronidase reshapes the extracellular matrix to enhance cancer chemotherapy and immunotherapy (7 March 2024) 

Oncolytic herpes simplex virus expressing IL-2 controls glioblastoma growth and improves survival  (9 April 2024) 

Challenges and opportunities in cancer immunotherapy: a Society for Immunotherapy of Cancer (SITC) strategic vision (19 June 2024) 

Expert consensus guidelines on management and best practices for tumor-infiltrating lymphocyte cell therapy  (29 February 2024) 

Biomarkers for response to TIL therapy: a comprehensive review (13 March 2024) 

Chronic immune-related adverse events arising from immune checkpoint inhibitors: an update  (4 July 2024) 

Don’t Forget to Check Out These Other Great  JITC Special Series:
The Role of Local Immunotherapy in the Treatment of Cancer (2026)
Cancer Immunotherapy in Understudied Populations  (2024-2025)
The Next Wave of Immuno-Oncology: A Roadmap from SITC (2024-2025) 
Computational Immuno-Oncology  (2023-2025) 


APC Discounts

As a way to thank the SITC members who work tirelessly to advance the science and improve the lives of cancer patients, SITC members who are first, last, or corresponding authors on JITC articles at the time of acceptance will receive discounted Article Processing Charges (APCs) . This discount is applied post-acceptance, at which point a discount code is shared with the corresponding author. Learn more .

JITC also offers full waivers for the full APC (100% discount of the APC) where all authors are based in low-income countries (see policy). Requests for waivers must be made prior to submission. For additional information regarding these discounts, as well as institutional arrangements and editor/reviewer discounts, view the journal's APC policy . Additional questions may be directed to JITC Editor@sitcancer.org.  

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